#Embolic_nephritis in #kidney of #cattle
📸 Dr. Mehrnoush Nabizadeh (Iran)
Embolic nephritis
Embolic lesions in the kidney cause no clinical signs unless they are extensive in which case toxemia followed by terminal uremia. Transitory periods during which proteinuria and pyuria occur may be observed if urine samples are examined at frequent intervals.
Causes:
Embolic suppurative nephritis or renal abscess may occur after any septicemia and bacteremia when bacteria lodge in renal tissues the origin of emboli may be: -
(A) Sporadic cases:
Valvular endocarditis in all species.
Suppurative lesions in uterus, udder, navel and peritoneal cavity of cattle.
(B) Associated with systemic infections such as: -
1. Shigellosis in foals.
2. Erysipelas in pigs.
Septicaemic or bacteraemia strangles in horses.
Pathogenesis: -
Localization of single bacterial cell or bacteria in small clumps in renal tissues development of embolic suppurative lesions.
Emboli block larger vessels than capillaries infarction, local ischemia in the affected portion, and if this infarction is small there is no clinical signs.
Clinical signs only develop when emboli are multiple and destroy much of renal parenchyma.
If the urine checked repeatedly, the sudden appearance of proteinuria, casts, and microscopic hematuria without other signs of renal disease it suggests infarction.
Clinical signs:
Embolic nephritis may pass without clinical signs due to insufficient renal damage.
Enlargement of the kidney may be palpated by rectal examination.
Fatal uremia and toxemia may be occurring.
Large infarcts may cause bouts of transient abdominal pain.
Diagnosis:
Clinical signs.
Clinical pathology as that of pyelonephritis accompanied with cystitis or urethritis.
P.M. lesions:
Enlargement of kidney with abscess formation.
Much fibrous tissue surrounding the lesions.
Treatment:
Sensitivity tests to chose proper antibiotics.
Antibiotics for about 7-10 days and may be injected with specific enzymes...
#embolic_nephriris
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